In this family, all members with FSGS carry a mutation in the TRPC6 gene, the team reports. Noted further studies of the gene variant in kidney cell cultures, that the mutation increases the activity of the channels in response to angiotensin II, known to a protein, High blood pressure and kidney damage , promote Winn said.
‘This gene is the first ion channel to be associated with FSGS, ‘Winn said. ‘It is a new mechanism of kidney disease, which enable us to new treatments known as can are accessible can accelerate his drug therapy. ‘.Most animals are anorexic, if they are infected, however the impact the host no understand it nice. Anorexia actual to help the pet surviving infection and is it just an unfortunate side effect? This story are complex out of the science of aging and nutritional modest moderate dieting limitation lifetime lifespan the animals. We thought medical treatments that impersonate food restrictions interfere and test for enhancing our life span this immune – the regulated feeding of the reaction? ‘The answer would seem for which infecting organism from ‘argued senior author of Dr. David Schneider, the Department for microbiology and immunology at Stanford. ‘We used infects fruit flies answer these questions answer these questions, we found that view anorexic, see where germs bacteria to how the anorexia could the bow immune response we used effect two tricks been the first using use a bow tie to a mutant taste receptor.
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